Epstein barr virus causes lymphoma

There are several oncoviruses, or viruses that causes cancer:. There is currently no vaccine for Epstein-Barr virus. Hepatitis B virus HBV is spread through infected blood, semen and other body fluids. Hepatitis B is a leading cause of liver cancer. The hepatitis B vaccine is recommended for all children and adults. Hepatitis C virus HCV is spread through infected blood.

There is no vaccine against hepatitis C, but it is highly treatable. Human immunodeficiency virus HIV is spread through infected semen, vaginal fluids, blood and breast milk. It can enable other oncoviruses to cause cancer. There is no vaccine against HIV. Human herpes virus 8 HHV-8 is related to Kaposi sarcoma in people who have a weakened immune system.

That includes patients with HIV. Human papillomavirus HPV has at least 12 strains that can cause cancer in men and women, including anal, cervical, penile , throat, vaginal and vulvar cancer.

Boys and girls age should get the HPV vaccine. It is spread through infected semen, vaginal fluids, blood and breast milk. The infection is rarely found in the United States. In the most severe cases, people lose their ability to write, speak, and walk, and even the average sufferer has a shorter life expectancy.

Some evidence has suggested a link between EBV and multiple sclerosis, which has included finding traces of EBV in the lesions caused by the disorder. This new research , published Thursday in Science, seems to have accomplished just that. The research team, led by scientists at the Harvard T.

Chan School of Public Health, was able to track the long-term health of about 10 million active duty military members, thanks to a year collaboration with the U. At the start of their service, these members have their blood screened for HIV and are screened regularly every two years afterward.

But even though the risk is higher than for people who haven't had mono, it is still very small about 1 in 1, Brothers and sisters of young people with this disease have a higher risk for HL.

The risk is very high for an identical twin of a person with HL. While other molecular abnormalities must be able to contribute to myc deregulation in the same way, EBV association in Burkitt's lymphoma is probably determined by the age at which EBV infection occurs being more likely when infection occurs in very young children and perhaps also by other infectious diseases that numerically influence the fraction, and predominant stage of differentiation and hence translocation breakpoint sites of immature B cells infected by EBV.

The presence of EBV in many such cells greatly increases the incidence rate of Burkitt's lymphoma, since one of the genetic lesions needed to deregulate c-myc is already present.

Abstract Recent investigations indicate that Burkitt's lymphoma consists of several subtypes, defined by their clinical and molecular features.